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ORAL PRESENTATION / TAM METİN SÖZLÜ SUNUM
defense enzymes CAT, GPX1, and GSH. These findings support the critical role of oxidative
stress in the pathophysiology of autism.
Contrary to expectations, infliximab administration did not have a beneficial effect
on oxidative stress markers; rather, it led to an increase in some parameters. This
suggests that infliximab may have potentially adverse effects on oxidative stress in the
temporal cortex within the experimental autism model. Further comprehensive studies
are warranted to more thoroughly investigate the effects of infliximab on autism
spectrum disorder and oxidative stress.
CONFLICT OF INTEREST
The authors declare that there is no conflict of interest.
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