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ORAL PRESENTATION / TAM METİN SÖZLÜ SUNUM



               defense enzymes CAT, GPX1, and GSH. These findings support the critical role of oxidative
               stress in the pathophysiology of autism.

                     Contrary to expectations, infliximab administration did not have a beneficial effect
               on  oxidative  stress  markers;  rather,  it  led  to  an  increase  in  some  parameters.  This

               suggests that infliximab may have potentially adverse effects on oxidative stress in the

               temporal cortex within the experimental autism model. Further comprehensive studies
               are  warranted  to  more  thoroughly  investigate  the  effects  of  infliximab  on  autism

               spectrum disorder and oxidative stress.


                                                CONFLICT OF INTEREST
                     The authors declare that there is no conflict of interest.



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