Page 49 - congress
P. 49
ORAL PRESENTATION / TAM METİN SÖZLÜ SUNUM
Bigler ED, Mortensen S, Neeley ES, Ozonoff S, Krasny L, Johnson M, et al. (2007). Superior
temporal gyrus, language function, and autism. Dev Neuropsychol. 31(2):217-38.
Bishayi B, Dey R, Ghosh R, Kanwar M. (2024). Simultaneous blockade of TLR4 and TNFR1
attenuates TLR2 sensitivity in LPS-stimulated macrophages through TNFR2-mediated
pathway. Indian J Physiol Allied Sci. 76(03):42-55.
Bjørklund G, Meguid NA, El-Bana MA, Tinkov AA, Saad K, Dadar M, Chirumbolo S. (2020). Oxidative
stress in autism spectrum disorder. Mol Neurobiol. 57(5):2314-2332.
Bruce AJ, Boling W, Kindy MS, Peschon J, Kraemer PJ, Carpenter MK, Mattson MP. (1996). Altered
neuronal and microglial responses to excitotoxic and ischemic brain injury in mice lacking
TNF receptors. Nat Med. 2(7):788-794.
Campara K, Rodrigues P, Viero FT, da Silva B, Trevisan G. (2025). A systematic review and meta-
analysis of advanced oxidative protein products levels (AOPP) levels in endometriosis:
Association with disease stage and clinical implications. Eur J Pharmacol. 177434.
Candel S, De Oliveira S, Lopez-Munoz A, Garcia-Moreno D, Espin-Palazon R, Tyrkalska SD, Mulero
V. (2014). Tnfa signaling through Tnfr2 protects skin against oxidative stress–induced
inflammation. PLoS Biol. 12(5):e1001855.
Chauhan A, Gu F, Essa MM, Wegiel J, Kaur K, Brown WT, Chauhan V. (2011). Brain region-specific
deficit in mitochondrial electron transport chain complexes in children with autism. J
Neurochem. 117(2):209-220.
Clarke JR, Lyra e Silva NM, Figueiredo CP, Frozza RL, Ledo JH, Beckman D, et al. (2015). Alzheimer-
associated Aβ oligomers impact the central nervous system to induce peripheral metabolic
deregulation. EMBO Mol Med. 7(2):190-210.
Dirik D, Kömüroğlu AU. (2021). The effect of infliximab on oxidative stress in ovarian tissue of the
rat with ovarian hyperstimulation syndrome. East J Med. 26(3).
Długosz A, Wróblewski M, Błaszak B, Szulc J. (2025). The role of nutrition, oxidative stress, and
trace elements in the pathophysiology of autism spectrum disorders. Int J Mol Sci.
26(2):808.
El-Ansary A, Bjørklund G, Chirumbolo S, Alnakhli OM. (2017). Predictive value of selected
biomarkers related to metabolism and oxidative stress in children with autism spectrum
disorder. Metab Brain Dis. 32(4):1209-1221.
Essa MM, Guillemin GJ, Waly MI, Al-Sharbati MM, Al-Farsi YM, Hakkim FL, Al-Shafaee MS. (2012).
Increased markers of oxidative stress in autistic children of the Sultanate of Oman. Biol
Trace Elem Res. 147(1):25-27.
Frustaci A, Neri M, Cesario A, Adams JB, Domenici E, Dalla Bernardina B, Bonassi S. (2012).
Oxidative stress-related biomarkers in autism: systematic review and meta-analyses. Free
Radic Biol Med. 52(10):2128-2141.
Giulivi C, Zhang YF, Omanska-Klusek A, Ross-Inta C, Wong S, Hertz-Picciotto I, Pessah IN. (2010).
Mitochondrial dysfunction in autism. JAMA. 304(21):2389-2396.
Habib R, Wahdan SA, Gad AM, Azab SS. (2019). Infliximab abrogates cadmium-induced testicular
damage and spermiotoxicity via enhancement of steroidogenesis and suppression of
inflammation and apoptosis mediators. Ecotoxicol Environ Saf. 182:109398.
Hellings J. (2023). Pharmacotherapy in autism spectrum disorders, including promising older
drugs warranting trials. World J Psychiatry. 13(6):262.
Hoshiyama T, Matsueda Y, Tono T, Arinuma Y, Nagai T, Hirohata S. (2018). Differential influences
of Fc gamma receptor blocking on the effects of certolizumab pegol and infliximab on
human monocytes. Modern Rheumatol. 28(3):506-512.
47

